Novel function of HATs and HDACs in homologous recombination through acetylation of human RAD52 at double-strand break sites

Journal: PLOS Genetics

Published: 2018-03-28

DOI: 10.1371/journal.pgen.1007277

Affiliations: 17

Authors: 28

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Institutions FC
Research Center for Radiation Protection, QST, Japan 0.30
National Institute of Radiological Sciences (NIRS), QST, Japan 0.11
Department of Interdisciplinary Science and Engineering, Meisei University, Japan 0.07
Department of Environmental and Radiological Health Sciences (ERHS), Colorado State University, United States of America (USA) 0.07
RIKEN Biomolecular Characterization Unit, Japan 0.07
International Open Laboratory (IOL), QST, Japan 0.07
Research Institute of Environmental Medicine (RIEM), Nagoya University, Japan 0.05
Research Center for Charged Particle Therapy, QST, Japan 0.05
Nuclear Technology Research Laboratory, CRIEPI, Japan 0.04
School of Life Science and Technology, Tokyo Tech, Japan 0.04
Faculty of Science, Gakushuin University, Japan 0.04
Department of Genome Repair, Nagasaki University, Japan 0.02
Cell-Bionomics Research Unit, KU, Japan 0.02
Biosignal Research Center, Kobe University, Japan 0.02
Faculty / Graduate School of Science, Kobe University, Japan 0.02
MRC Genome Damage and Stability Centre (GDSC), University of Sussex, United Kingdom (UK) 0.02

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