Clonally expanding smooth muscle cells promote atherosclerosis by escaping efferocytosis and activating the complement cascade.

Institutions Share
Stanford Division of Vascular Surgery, United States of America (USA) 0.44
Stanford Cardiovascular Institute (CVI), United States of America (USA) 0.08
Robert M. Berne Cardiovascular Research Center (CVRC), UVA, United States of America (USA) 0.08
Department of Medical Sciences, UU, Sweden 0.05
Icahn Institute for Genomics and Multiscale Biology, ISMMS, United States of America (USA) 0.04
Department of Genetics and Genomic Sciences, ISMMS, United States of America (USA) 0.04
Stanford Institute for Stem Cell Biology and Regenerative Medicine (ISCBRM), United States of America (USA) 0.04
Stanford Division of Pediatric Cardiology, United States of America (USA) 0.04
Tartu University Hospital, UT, Estonia 0.04
University Medical Center Utrecht (UMC Utrecht), Netherlands 0.02
UMC Laboratory of Clinical Chemistry and Haematology (LKCH), Netherlands 0.02
TUM University Hospital Klinikum rechts der Isar (MRI), Germany 0.02
Stanford Department of Bioengineering, United States of America (USA) 0.02
DZHK Partner Site Munich, Germany 0.02
UAB School of Medicine (SOM), United States of America (USA) 0.01
Stanford Division of Cardiovascular Medicine, United States of America (USA) 0.01
Department of Public Health Sciences, UVA, United States of America (USA) 0.01
Center for Public Health Genomics (CPHG), UVA, United States of America (USA) 0.01
Department of Biochemistry and Molecular Genetics (BMG), UVA, United States of America (USA) 0.01
Department of Biomedical Engineering (BME), UVA, United States of America (USA) 0.01

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