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Endogenous FGF21-signaling controls paradoxical obesity resistance of UCP1-deficient mice

Journal: Nature Communications

Published: 2020-01-31

DOI: 10.1038/s41467-019-14069-2

Affiliations: 11

Authors: 12

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Institutions Share
TUM-HMGU Institute of Human Genetics (IHG), Germany 0.17
German Center for Diabetes Research (DZD), Germany 0.17
Institute for Diabetes and Obesity (IDO), HMGU, Germany 0.12
Helmholtz Diabetes Center (HDC), HMGU, Germany 0.12
German Mouse Clinic (GMC), HMGU, Germany 0.09
Institute of Experimental Genetics (IEG), HMGU, Germany 0.09
Wallenberg Laboratory for Cardiovascular and Metabolic Research, GU, Sweden 0.08
Institute of Medicine, GU, Sweden 0.08
Department of Molecular Biosciences / The Wenner-Gren Institute (MBW), Stockholm University, Sweden 0.04
TUM Chair of Experimental Genetics, Germany 0.02
TUM Chair of Metabolic Diseases, Germany 0.02

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