The structure of the ubiquitin-like modifier FAT10 reveals an alternative targeting mechanism for proteasomal degradation

Journal: Nature Communications

Published: 2018-08-20

DOI: 10.1038/s41467-018-05776-3

Affiliations: 5

Authors: 14

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Institutions FC
Max Planck Institute for Developmental Biology (MPI-EB), Germany 0.29
Biotechnology Institute Thurgau (BITg), Switzerland 0.29
Department of Biology, University of Konstanz, Germany 0.21
Department of Chemistry, University of Konstanz, Germany 0.14
Institute of Biophysics and Physical Biochemistry, UR, Germany 0.07